Ectromelia virus (ECTV) is a virus of the family Poxviridae and the genus Orthopoxvirus that causes mousepox, a disease of mice. It has only been seen in mouse colonies kept for research purposes but believed that wild populations of mice and other rodents in Europe are naturally infected with ECTV. [1][2] Mousepox causes skin lesions, a ...

Etiology: Ectromelia virus, the agent of mousepox, is an enveloped DNA poxvirus of the vaccinia subgroup. Incidence: Incidence of disease is rare, with sporadic epizootics usually resulting from passage of infected cells or other biological material into naive mice.

Ectromelia is a congenital condition where long bones are missing or underdeveloped. [1] Examples include: Amelia Hemimelia Phocomelia Sirenomelia

Ectromelia virus (ECTV) is an orthopoxvirus that is a close relative of VARV and VACV and is a natural pathogen to mice, causing mousepox [29]. From: Trends in Immunology, 2005

A disease of mice caused by the ectromelia virus, a member of the family Poxviridae; characterized by gangrenous loss of feet and by necrotic areas in the internal organs; in laboratory mouse colonies, it usually results in high mortality rates.

Ectromelia virus (ECTV) is an orthopoxvirus whose natural host is the mouse; it is related closely to Variola virus, the causative agent of smallpox, and Monkeypox virus, the cause of an emerging zoonosis.

The meaning of ECTROMELIA is congenital absence or imperfection of one or more limbs.

Ectromelia virus, which causes the disease mousepox, is transmitted by direct contact or by fomites. Although many routes of infection are possible experimentally, exposure to the virus via cutaneous trauma is the natural route of infection.

2003年12月5日 · Ectromelia virus (ECTV) is the causative agent of mousepox, an acute exanthematous disease of mouse colonies in Europe, Japan, China, and the U.S. Buller and Palumbo, 1991, Fenner, 1982. The disease is on the decline in vivariums due to improvements in animal husbandry and health surveillance.

Ectromelia virus lacking the E3L ortholog is replication-defective and nonpathogenic but does induce protective immunity in a mouse strain susceptible to lethal mousepox.