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肝癌耐药新发现!中山大学合作发文:揭示肝癌中索拉非尼耐药关键 ...
从机制上讲,由索拉非尼引发的活性氧 - 内质网应激轴触发的 TRIB3 以 NF-κB 依赖的方式发挥作用,上调 CXCR1/2 配体,随后促进中性粒细胞向肿瘤募集。
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