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生物通
3 年
新型慢性胰腺炎动物模型解读
例如,PRSS1(阳离子胰蛋白酶原)、CTRC(胰凝乳蛋白酶C)以及SPINK1(Kazal型丝氨酸蛋白酶抑制剂1)的致病突变通过刺激自发激活或干扰保护机制,从而促进胰蛋白酶原转化成有害的胰蛋白酶。此外,慢性胰腺炎的遗传风险也可以通过与胰蛋白酶活性无关的机制来 ...
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