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无氧代谢通过组蛋白-3赖氨酸-18乳酸化介导PPARD轴促进乳腺癌存活
研究人员运用多种实验技术,发现乳腺癌组织中 H3K18la 水平高于癌旁组织。H3K18la 促进 PPARD 表达,进而影响 AKT 转录,且糖酵解增强了乳腺癌细胞的染色质可及性。同时证实 HDAC2 和 HDAC3 是 H3 赖氨酸乳酸化的 “擦除器”。该研究表明,乳腺癌中 H3K18la 及其下游 ...
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